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Selank in T1D Research: Stress Biology and Immune Modulation HPA Axis Dysregulation in T1D T1D is associated with HPA axis dysregulation — elevated cortisol in early-onset T1D and abnormal CRH-ACTH dynamics — that amplifies the Th1 response through glucocorticoid receptor downregulation and impaired Treg sensitivity to cortisol-driven FoxP3 induction
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